,
,
,
,
,
,
Open Access
Article Info
- Michele Columbo and
- Albert S. Rohr
April 13, 2016
April 12, 2016
Asthma in furniture and wood processing workers: a systematic review
R. E. Wiggans1,2,
G. Evans3,
D. Fishwick1,2 and
C. M. Barber1,2
+Author Affiliations
Abstract
Background Wood dust is a common cause of occupational asthma. There is potential for high exposure to wood dust during furniture and wood manufacturing processes.
Aims To evaluate the evidence for non-neoplastic respiratory ill health associated with work in the furniture and wood manufacturing sector.
April 11, 2016
Asthma in the elderly: the effect of choline supplementation
Abstract
Background
Asthma in the elderly is poorly understood as very few studies have included these patients. DNA methylation can affect the expression of asthma susceptibility genes. Methyl groups can be produced through a choline dependent pathway. Asthmatics have decreased serum choline. We studied the effect of choline supplementation in elderly asthmatics and associations between different parameters at baseline.
The cytokine interleukin-26 as a biomarker in pediatric asthma
LETTER TO THE EDITOR
OPEN ACCESS
Abstract
In this pilot study, we examined associations between local interleukin (IL)-26, disease severity and biomarkers of Th2-mediated inflammation in a well-defined cohort of pediatric patients (14 years median age, 41 % females) with controlled (n = 28) or uncontrolled (n = 48) asthma.
Detailed analysis of sputum and systemic inflammation in asthma phenotypes: are paucigranulocytic asthmatics really non-inflammatory?
DOI: 10.1186/s12890-016-0208-2
© Demarche et al. 2016
Abstract
Background
The technique of induced sputum has allowed to subdivide asthma patients into inflammatory phenotypes according to their level of granulocyte airway infiltration. There are very few studies which looked at detailed sputum and blood cell counts in a large cohort of asthmatics divided into inflammatory phenotypes. The purpose of this study was to analyze sputum cell counts, blood leukocytes and systemic inflammatory markers in these phenotypes, and investigate how those groups compared with healthy subjects.
April 10, 2016
Eosinophilic esophagitis–linked calpain 14 is an IL-13–induced protease that mediates esophageal epithelial barrier impairment
April 9, 2016
Nasal polyps in patients with asthma: prevalence, impact, and management challenges
Authors Langdon C, Mullol J
Received 1 October 2015
Accepted for publication 5 January 2016
Published 14 March 2016 Volume 2016:9 Pages 45—53
Cristobal Langdon,1,2 Joaquim Mullol1–3
1Rhinology Unit and Smell Clinic, Otorhinolaryngology Department, Hospital Clínic, 2Clinical and Experimental Respiratory Immunoallergy (IRCE), Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS), 3Centre for Biomedical Research in Respiratory Diseases (CIBERES), Barcelona, Catalonia, Spain
Abstract: Patients with chronic rhinosinusitis with nasal polyps (CRSwNP) often have coexisting asthma under the concept of “United Airway Disease”, being the combination of both diseases, which is one of the most challenging phenotypes to treat.
1Rhinology Unit and Smell Clinic, Otorhinolaryngology Department, Hospital Clínic, 2Clinical and Experimental Respiratory Immunoallergy (IRCE), Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS), 3Centre for Biomedical Research in Respiratory Diseases (CIBERES), Barcelona, Catalonia, Spain
Abstract: Patients with chronic rhinosinusitis with nasal polyps (CRSwNP) often have coexisting asthma under the concept of “United Airway Disease”, being the combination of both diseases, which is one of the most challenging phenotypes to treat.
Subscribe to:
Posts (Atom)